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Secoisolariciresinol, a natural phenolic compound, is derived from the heartwood of the Mongolian pine tree (Pinus sylvestris). It exhibits a range of biological activities, including antioxidant, anti-inflammatory, and antimicrobial properties. Its unique structure, featuring two phenolic rings connected by a dihydrofuran ring, contributes to its diverse pharmacological effects.

29388-59-8

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29388-59-8 Usage

Uses

Used in Pharmaceutical Industry:
Secoisolariciresinol is used as a therapeutic agent for its anti-myocardial ischemia effects, which can help in the treatment of heart diseases by reducing the damage caused by ischemic conditions.
Used in Antioxidant Applications:
Secoisolariciresinol is used as an antioxidant, protecting cells from oxidative stress and damage caused by reactive oxygen species. Its antioxidant properties can be beneficial in various health and skincare products, as well as in the food industry to extend the shelf life of products.
Used in Antimicrobial Applications:
Secoisolariciresinol is used as an antimicrobial agent, exhibiting activity against a range of bacteria and fungi. This makes it a potential candidate for use in the development of new antimicrobial drugs and in the formulation of natural preservatives for various applications.
Used in Mongolian Folk Medicine:
Secoisolariciresinol is used as a traditional medicine in Mongolia, where it has been employed for centuries to treat various ailments, including heart diseases and infections, due to its multifaceted health-promoting properties.

Check Digit Verification of cas no

The CAS Registry Mumber 29388-59-8 includes 8 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 5 digits, 2,9,3,8 and 8 respectively; the second part has 2 digits, 5 and 9 respectively.
Calculate Digit Verification of CAS Registry Number 29388-59:
(7*2)+(6*9)+(5*3)+(4*8)+(3*8)+(2*5)+(1*9)=158
158 % 10 = 8
So 29388-59-8 is a valid CAS Registry Number.
InChI:InChI=1/C20H26O6/c1-25-19-9-13(3-5-17(19)23)7-15(11-21)16(12-22)8-14-4-6-18(24)20(10-14)26-2/h3-6,9-10,15-16,21-24H,7-8,11-12H2,1-2H3/t15-,16-/m0/s1

29388-59-8SDS

SAFETY DATA SHEETS

According to Globally Harmonized System of Classification and Labelling of Chemicals (GHS) - Sixth revised edition

Version: 1.0

Creation Date: Aug 19, 2017

Revision Date: Aug 19, 2017

1.Identification

1.1 GHS Product identifier

Product name (-)-secoisolariciresinol

1.2 Other means of identification

Product number -
Other names (2R*,3R*)-2,3-Bis(4-hydroxy-3-methoxybenzyl)-1,4-butanediol

1.3 Recommended use of the chemical and restrictions on use

Identified uses For industry use only.
Uses advised against no data available

1.4 Supplier's details

1.5 Emergency phone number

Emergency phone number -
Service hours Monday to Friday, 9am-5pm (Standard time zone: UTC/GMT +8 hours).

More Details:29388-59-8 SDS

29388-59-8Relevant academic research and scientific papers

Antiestrogenic and antiproliferative potency of secoisolariciresinol diglucoside derivatives on MCF-7 breast cancer cells

Scherbakov, Alexander M.,Stasevich, Olga V.,Salnikova, Diana I.,Andreeva, Olga E.,Mikhaevich, Ekaterina I.

supporting information, p. 6099 - 6105 (2020/10/12)

Secoisolariciresinol diglucoside (SDG) is isolated from Linum usitatissimum seeds. The antiproliferative effects of SDG (1) and its derivatives secoisolariciresinol (2) and secoisolariciresinol-4′, 4″-diacetate (3) have been evaluated on MCF-7 breast cancer cells and normal breast epithelial line MCF-10A. Lignan 1 has not shown cytotoxic effects on MCF-7 cells, while derivatives 2 and 3 have inhibited cell growth with IC50 values of 25 and 11 μM, respectively. Estrogen receptor alpha is a key growth driver in MCF-7 cells. Compound 1 did not affect the activity of ERα, while derivatives 2 and 3 showed significant antiestrogenic effects. Compounds 2 and 3 caused apoptosis in the MCF-7 line, determined by the cleavage of PARP. SDG derivative 3 enhanced the effect of doxorubicin. SDG derivatives can be considered as promising agents that exhibit a combined antiestrogen and proapoptotic effect in hormone-dependent breast cancer cells.

Total Synthesis and Stereochemical Confirmation of (-)-Olivil, (+)-Cycloolivil, (-)-Alashinols F and G, (+)-Cephafortin A, and Their Congeners: Filling in Biosynthetic Gaps

Hanessian, Stephen,Reddy Vakiti, Jithender

supporting information, p. 3345 - 3350 (2020/04/30)

For the first time, we describe the stereocontrolled total syntheses of olivil, cephafortin A, 4-des-O-methyl-4-O-rhamnosyl cephafortin A, and alashinol F from a common precursor using a combination of chemoenzymatic and biomimetic methods for the systematic introduction of functional groups on three vicinal stereogenic carbon atoms. We revised the previously assigned stereochemistry of (+)-cephafortin A, which was reported as the enantiomer. Natural and unnatural congeners provide insights into the biogenetic interrelations of members of this family.

Pinoresinol-lariciresinol reductase: Substrate versatility, enantiospecificity, and kinetic properties

Davin, Laurence B.,Hwang, Julianne K.,Lewis, Norman G.,Moinuddin, Syed G. A.

, (2020/03/26)

Two western red cedar pinoresinol-lariciresinol reductase (PLR) homologues were studied to determine their enantioselective, substrate versatility, and kinetic properties. PLRs are downstream of dirigent protein engendered, coniferyl alcohol derived, stereoselective coupling to afford entry into the 8- and 8′-linked furofuran lignan, pinoresinol. Our investigations showed that each PLR homolog can enantiospecifically metabolize different furofuran lignans with modified aromatic ring substituents, but where phenolic groups at both C4/C4′ are essential for catalysis. These results are consistent with quinone methide intermediate formation in the PLR active site. Site-directed mutagenesis and kinetic measurements provided additional insight into factors affecting enantioselectivity and kinetic properties. From these data, PLRs can be envisaged to allow for the biotechnological potential of generation of various lignan skeleta, that could be differentially “decorated” on their aromatic ring substituents, via the action of upstream dirigent proteins.

Modification method for improving oil solubility of lignan

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Paragraph 0048-0051, (2019/10/08)

The invention belongs to the food field and particularly relates to a modification method for improving oil solubility of lignan. The modification method comprises the step of modifying glycosyl of the lignan, including esterification modification of the glycosyl of the lignan with a chemical method or an enzyme method or hydrolysis modification of the lignan with the enzyme method to remove the glycosyl of the lignan, a lignan derivative containing fewer hydroxyls is produced, so that the lipid solubility of the lignan is improved. The lipid solubility of the lignan is improved, and the obtained product has the advantages that the product can be applied to food and medicine conveniently and can reduce dosage of additional additives, particularly emulsifiers; the esterification product can be used as an emulsifier to be applied to the fields of food and the like and has healthcare functions of lignan and fatty acid. High-value application of the lignan is increased with those lignan modification methods.

Pharmaceutical compositions comprising 8-substituted dibenzylbutyrolactone lignans

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Page/Page column 10, (2015/11/09)

Therapeutic compositions comprising at least one 8-substituted-dibenzylbutyrolactone lignan, preferably a lignan is selected from the group of nortrachelogenin, diasteromeric forms of nortrachelogenin, isomeric forms of nortrachelogenin and combinations thereof as well as 8-methylmatairesinol and 8-methyldimethylmatairesinol, for use in a method of treating cancer or a similar condition wherein the growth factor signaling pathway of a mammal is deregulated. The invention also provides therapeutic pharmaceutical combinations comprising a hydroxy-dibenzylbutyrolactone lignan and at least one TRAIL receptor agonist. The hydroxy-dibenzylbutyrolactone lignans and a TRAIL receptor agonist can be used as a combined preparation for administration to a patient simultaneously, separately or spaced out over a period of time in treating cancer.

Ring substitution influences oxidative cyclisation and reactive metabolite formation of nordihydroguaiaretic acid analogues

Asiamah, Isaac,Hodgson, Heather L.,Maloney, Katherine,Allen, Kevin J.H.,Krol, Ed S.

supporting information, p. 7007 - 7014 (2015/11/11)

Nordihydroguaiaretic acid (NDGA) is a natural polyphenol with a broad spectrum of pharmacological properties. However, its usefulness is hindered by the lack of understanding of its pharmacological and toxicological pathways. Previously we showed that oxidative cyclisation of NDGA at physiological pH forms a dibenzocyclooctadiene that may have therapeutic benefits whilst oxidation to an ortho-quinone likely mediates toxicological properties. NDGA analogues with higher propensity to cyclise under physiologically relevant conditions might have pharmacological implications, which motivated this study. We synthesized a series of NDGA analogues which were designed to investigate the structural features which influence the intramolecular cyclisation process and help to understand the mechanism of NDGA's autoxidative conversion to a dibenzocyclooctadiene lignan. We determined the ability of the NDGA analogues investigated to form dibenzocyclooctadienes and evaluated the oxidative stability at pH 7.4 of the analogues and the stability of any dibenzocyclooctadienes formed from the NDGA analogues. We found among our group of analogues the catechols were less stable than phenols, a single catechol-substituted ring is insufficient to form a dibenzocyclooctadiene lignan, and only compounds possessing a di-catechol could form dibenzocyclooctadienes. This suggests that quinone formation may not be necessary for cyclisation to occur and the intramolecular cyclisation likely involves a radical-mediated rather than an electrophilic substitution process. We also determined that the catechol dibenzocyclooctadienes autoxidised at comparable rates to the parent catechol. This suggests that assigning in vitro biological activity to the NDGA dibenzocyclooctadiene is premature and requires additional study.

An access to chiral β-benzyl-γ-butyrolactones and its application to the synthesis of enantiopure (+)-secoisolariciresinol, (-)-secoisolariciresinol, and (-)-enterolactone

Allais, Florent,Pla, Thomas J. L.,Ducrot, Paul-Henri

, p. 1456 - 1464 (2011/06/17)

Both enantiomers of secoisolariciresinol and enantiopure (-)-enterolactone were synthesized through a highly stereoselective convergent synthesis. An Evans diastereoselective alkylation followed by a substrate-induced diastereoselective -alkylation of the newly formed optically active β-benzyl-γ- butyrolactone gave the β-β′ linkage of the target skeleton. The (S,S)- and (R,R)-enantiomers of secoisolariciresinol and (-)-enterolactone were obtained in 12-14% (11 steps) and 20% (7 steps) overall yield, respectively. Georg Thieme Verlag Stuttgart New York.

Synthesis and evaluation of cytotoxic effects of hanultarin and its derivatives

Lee, Eunyoung,Jamal Ahamed,Kumar, Mahto Sanjeev,Rhee, Seog Woo,Moon, Surk-Sik,Hong, In Seok

supporting information; experimental part, p. 6245 - 6248 (2011/12/02)

One of the known cytotoxic lignans is (-)-1-O-feruloyl-secoisolariciresinol designated as hanultarin, which was isolated from the seeds of Trichosanthes kirilowii. In this Letter, we described the first synthesis of 1-O-feruloyl-secoisolariciresinol, 1,4-O-diferuloyl-secoisolariceresinol and their analogues. The cytotoxicities of these compounds were evaluated against several cancer cell lines. Interestingly, we found that the feruloyl diester derivative of secoisolariciresinol was the most active cytotoxic compound against all the cancer cells tested in this experiment. The IC50 values of the1,4-O-diferuloyl-secoisolariceresinol were in the range of 7.1-9.8 lM except one cell line. In considering that both ferulic acid and secoisolariciresinol are commonly found in many plants and have no cytotoxicity, this finding is remarkable in that simple covalent bonds between the ferulic acid and secoisolariciresinol cause a cytotoxic effect.

Total synthesis of (±)-agastinol

Ding, Junwei,Zhou, Haitang,Jiao, Bin,Xia, Yamu

scheme or table, p. 352 - 354 (2011/10/08)

A synthesis of the tetrahydrofuran lignan (±)-agastinol, starting from the cheap Vanillin, has been developed based on Stobbe reaction with diethyl succinate to give the skeleton of lignan, which was then reduced to afford meso- and threo-(±)-secoisolanciresinol. threo-(±)- Secoisolanciresinol was treated with DDQ in acetic acid to give the 2-aryl tetrahydrofuran lignan, and which was then condensed with ferulic acid to give (±)-agastinol for the first time.

Synthesis of threo-(±)-9,9-dibenzoylsecoisolariciresinol and its isomer

Xia, Yamu,Wen, Yanling

experimental part, p. 606 - 609 (2011/02/28)

The total synthesis of threo-(±)-9,9-dibenzoylsecoisolariciresinol and its isomer based on two Stobbe reactions as C-C bond-forming steps used a protected vanillin and diethyl; succinate to give the skeleton of lignan, followed by reduction to afford threo-and meso-(±)-secoisolariciresinol Both were treated with benzoyl chloride to obtain the natural product threo-(±)-9,9-dibenzoylsecoisolariciresinol and Its isomer meso-(±)-9,9-dibenzoylsecoisolariciresinol for the first time.

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