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benzyl 3-(benzyloxy)benzoate is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

54396-39-3

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54396-39-3 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 54396-39-3 includes 8 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 5 digits, 5,4,3,9 and 6 respectively; the second part has 2 digits, 3 and 9 respectively.
Calculate Digit Verification of CAS Registry Number 54396-39:
(7*5)+(6*4)+(5*3)+(4*9)+(3*6)+(2*3)+(1*9)=143
143 % 10 = 3
So 54396-39-3 is a valid CAS Registry Number.

54396-39-3SDS

SAFETY DATA SHEETS

According to Globally Harmonized System of Classification and Labelling of Chemicals (GHS) - Sixth revised edition

Version: 1.0

Creation Date: Aug 19, 2017

Revision Date: Aug 19, 2017

1.Identification

1.1 GHS Product identifier

Product name benzyl 3-(benzyloxy)benzoate

1.2 Other means of identification

Product number -
Other names -

1.3 Recommended use of the chemical and restrictions on use

Identified uses For industry use only.
Uses advised against no data available

1.4 Supplier's details

1.5 Emergency phone number

Emergency phone number -
Service hours Monday to Friday, 9am-5pm (Standard time zone: UTC/GMT +8 hours).

More Details:54396-39-3 SDS

54396-39-3Relevant articles and documents

How Many O-Donor Groups in Enterobactin Does It Take to Bind a Metal Cation?

Baramov, Todor,Schmid, Bianca,Ryu, Ho,Jeong, Jinhoon,Keijzer, Karlijn,von Eckardstein, Leonard,Baik, Mu-Hyun,Süssmuth, Roderich D.

supporting information, p. 6955 - 6962 (2019/04/17)

The E. coli siderophore enterobactin, the strongest FeIII chelator known to date, forms hexacoordinate complexes with SiIV, GeIV, and TiIV. Synthetic protocols have been developed to prepare non-symmetric entero

HIF-1α inhibitors: Synthesis and biological evaluation of novel moracin O and P analogues

Xia, Yan,Jin, Yinglan,Kaur, Navneet,Choi, Yongseok,Lee, Kyeong

supporting information; experimental part, p. 2386 - 2396 (2011/06/21)

The natural products moracins O and P exhibited potent in vitro inhibitory activity against hypoxia-inducible factor (HIF-1), which is a key mediator during adaptation of cancer cells to tumour hypoxia. Systematic variations of the structures of benzofuran type moracins were made and structure-activity relationship analysis showed the importance of the 2-arylbenzofuran ring and the (R)-configuration of the core scaffold. Further evaluation of the representative compound 5 showed its inhibitory effect on HIF-1α protein accumulation and target gene expression under hypoxia.

COMPOSITIONS AND METHODS FOR GLUCOSE TRANSPORT INHIBITION

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Page/Page column 27-28, (2011/10/13)

Glucose deprivation is an attractive strategy in cancer research and treatment. Cancer cells upregulate glucose uptake and metabolism for maintaining accelerated growth and proliferation rates. Specifically blocking these processes is likely to provide new insights to the role of glucose transport and metabolism in tumorigenesis, as well as in apoptosis. As solid tumors outgrow the surrounding vasculature, they encounter microenvironments with a limited supply of nutrients leading to a glucose deprived environment in some regions of the tumor. Cancer cells living in the glucose deprived environment undergo changes to prevent glucose deprivation-induced apoptosis. Knowing how cancer cells evade apoptosis induction is also likely to yield valuable information and knowledge of how to overcome the resistance to apoptosis induction in cancer cells. Disclosed herein are novel anticancer compounds that inhibit basal glucose transport, resulting in tumor suppression and new methods for the study of glucose deprivation in animal cancer research.

Novel inhibitors of basal glucose transport as potential anticancer agents

Zhang, Weihe,Liu, Yi,Chen, Xiaozhuo,Bergmeier, Stephen C.

scheme or table, p. 2191 - 2194 (2010/06/15)

Cancer cells commonly show increased levels of glucose uptake and dependence. A potential strategy for the treatment of cancer may be the inhibition of basal glucose transport. We report here the synthesis of a small library of polyphenolic esters that inhibit basal glucose transport in H1299 lung and other cancer cells. These basal glucose transport inhibitors also inhibit cancer cell growth in H1299 cells, and these two activities appear to be correlated.

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