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1-ethoxy-4-(isothiocyanatomethyl)benzene is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

18636-22-1

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18636-22-1 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 18636-22-1 includes 8 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 5 digits, 1,8,6,3 and 6 respectively; the second part has 2 digits, 2 and 2 respectively.
Calculate Digit Verification of CAS Registry Number 18636-22:
(7*1)+(6*8)+(5*6)+(4*3)+(3*6)+(2*2)+(1*2)=121
121 % 10 = 1
So 18636-22-1 is a valid CAS Registry Number.

18636-22-1Downstream Products

18636-22-1Relevant academic research and scientific papers

Structure-Activity Relationships and Computational Investigations into the Development of Potent and Balanced Dual-Acting Butyrylcholinesterase Inhibitors and Human Cannabinoid Receptor 2 Ligands with Pro-Cognitive in Vivo Profiles

Dolles, Dominik,Hoffmann, Matthias,Gunesch, Sandra,Marinelli, Oliviero,M?ller, Jan,Santoni, Giorgio,Chatonnet, Arnaud,Lohse, Martin J.,Wittmann, Hans-Joachim,Strasser, Andrea,Nabissi, Massimo,Maurice, Tangui,Decker, Michael

, p. 1646 - 1663 (2018)

The enzyme butyrylcholinesterase (BChE) and the human cannabinoid receptor 2 (hCB2R) represent promising targets for pharmacotherapy in the later stages of Alzheimer's disease. We merged pharmacophores for both targets into small benzimidazole-based molecules, investigated SARs, and identified several dual-acting ligands with a balanced affinity/inhibitory activity and an excellent selectivity over both hCB1R and hAChE. A homology model for the hCB2R was developed based on the hCB1R crystal structure and used for molecular dynamics studies to investigate binding modes. In vitro studies proved hCB2R agonism. Unwanted μ-opioid receptor affinity could be designed out. One well-balanced dual-acting and selective hBChE inhibitor/hCB2R agonist showed superior in vivo activity over the lead CB2 agonist with regards to cognition improvement. The data shows the possibility to combine a small molecule with selective and balanced GPCR-activity/enzyme inhibition and in vivo activity for the therapy of AD and may help to rationalize the development of other dual-acting ligands.

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