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(S)-2-[4-(2,4-Diamino-5,8-dihydro-6H-pyrido[4',3':4,5]furo[2,3-d]pyrimidin-7-ylmethyl)-benzoylamino]-pentanedioic acid diethyl ester is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

204995-46-0

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204995-46-0 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 204995-46-0 includes 9 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 6 digits, 2,0,4,9,9 and 5 respectively; the second part has 2 digits, 4 and 6 respectively.
Calculate Digit Verification of CAS Registry Number 204995-46:
(8*2)+(7*0)+(6*4)+(5*9)+(4*9)+(3*5)+(2*4)+(1*6)=150
150 % 10 = 0
So 204995-46-0 is a valid CAS Registry Number.

204995-46-0Downstream Products

204995-46-0Relevant academic research and scientific papers

Synthesis and biological activities of tricyclic conformationally restricted tetrahydropyrido annulated furo[2,3-d]pyrimidines as inhibitors of dihydrofolate reductases

Gangjee, Aleem,Elzein, Elfatih,Queener, Sherry F.,McGuire, John J.

, p. 1409 - 1416 (1998)

The synthesis of seven 2,4-diamino-5,6,7,8-tetrahydro-7-substituted pyrido[4',3':4,5]furo[2,3-d]pyrimidines 1-6 are reported as nonclassical antifolate inhibitors of dihydrofolate reductase (DHFR) and compound 7 as a classical antifolate inhibitor of tumor cells in culture. The compounds were designed as conformationally restricted analogues of trimetrexate. The synthesis was accomplished from the cyclocondensation of 3-bromo-4-piperidone with 2,4-diamino-6-hydroxypyrimidine to afford regiospecifically 2,4-diamino- 5,6,7,8-tetrahydropyrido-[4',3':4,5]furo[2,3-d]pyrimidine-7-hydrobromide (16). This in turn was alkylated with the appropriate benzyl halide to afford the target compounds 1-6. The classical antifolate 7 utilized 4- (chloromethyl)benzoyl-L-glutamic acid diethyl ester (17) instead of the benzyl halide for alkylation, followed by saponification to afford 7. Compounds 1-6 showed moderate inhibitory potency against DHFR from Pneumocystis carinii, Toxoplasma gondii, Mycobacterium avium, and rat liver. The classical analogue 7 was 88-fold more potent against M. avium DHFR than against rat liver DHFR. The classical analogue was also inhibitory against the growth of tumor cells, CCRF-CEM, and FaDu, in culture.

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