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1-(3,4-Dimethoxyphenyl)-3-(methoxycarbonyl)-1,2,3,4-tetrahydro-β-carboline is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

82789-36-4

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82789-36-4 Usage

Chemical class

β-carbolines

Explanation

1-(3,4-Dimethoxyphenyl)-3-(methoxycarbonyl)-1,2,3,4-tetrahydro-β-carboline belongs to a class of alkaloids known as β-carbolines, which are naturally occurring in various plants.

Explanation

It is a derivative of the naturally occurring alkaloid harmine, which is found in plants such as Banisteriopsis caapi and Peganum harmala.

Explanation

Studies have been conducted to explore the compound's potential pharmacological activities, particularly its effects on the central nervous system.

Explanation

The compound has been investigated for its potential use in treating various neurological and psychiatric disorders due to its modulatory effects on biological processes.

Explanation

Research has been conducted to determine if 1-(3,4-Dimethoxyphenyl)-3-(methoxycarbonyl)-1,2,3,4-tetrahydro-β-carboline can act as an antioxidant, which may help protect cells from damage caused by reactive oxygen species.

Explanation

The compound has been studied for its potential anti-inflammatory properties, which could be beneficial in treating conditions characterized by inflammation.

Explanation

1-(3,4-Dimethoxyphenyl)-3-(methoxycarbonyl)-1,2,3,4-tetrahydro-β-carboline is of interest due to its potential to modulate various biological processes, which may contribute to its therapeutic properties.

Derivative of

Harmine

Pharmacological activities

Potential effects on the central nervous system

Therapeutic potential

Treatment of neurological and psychiatric disorders

Antioxidant properties

Investigated for its potential role as an antioxidant

Anti-inflammatory properties

Investigated for its potential role as an anti-inflammatory agent

Modulation of biological processes

Ability to modulate various biological processes

Check Digit Verification of cas no

The CAS Registry Mumber 82789-36-4 includes 8 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 5 digits, 8,2,7,8 and 9 respectively; the second part has 2 digits, 3 and 6 respectively.
Calculate Digit Verification of CAS Registry Number 82789-36:
(7*8)+(6*2)+(5*7)+(4*8)+(3*9)+(2*3)+(1*6)=174
174 % 10 = 4
So 82789-36-4 is a valid CAS Registry Number.

82789-36-4Relevant academic research and scientific papers

β-Carboline tethered cinnamoyl 2-aminobenzamides as class I selective HDAC inhibitors: Design, synthesis, biological activities and modelling studies

Godugu, Chandraiah,Kamal, ahmed,Lakshmi Manasa, Kesari,Namballa, Hari Krishna,Shankaraiah, Nagula,Soni, Jay Prakash,anchi, Pratibha

, (2021/11/11)

The effect of β-carboline motif as cap for HDAC inhibitors containing cinnamic acid as linker and benzamides as zinc binding group was examined in this study. A series of β-carboline-cinnamide conjugates have been synthesized and evaluated for their HDAC inhibitory activity and in vitro cytotoxicity against different human cancer cell lines. Almost all the compounds exhibited superior HDAC inhibitory activity than the standard drug Entinostat for in vitro enzymatic assay. Among the tested compounds, 7h displayed a noteworthy potency with an IC50 value of 0.70 ± 0.15 μM against HCT-15 cell line when compared to the standard drug Entinostat (IC50 of 3.87 ± 0.62 μM). The traditional apoptosis assays such as nuclear morphological alterations, AO/EB, DAPI, and Annexin-V/PI staining revealed the antiproliferative activity of 7h while depolarization of mitochondrial membrane potential by JC-1 was observed in dose-dependent manner. Cell cycle analysis also unveiled the typical accumulation of cells in G2M phase and sub-G1/S phase arrest. In addition, immunoblot analysis for compound 7h on HCT-15 indicated selective inhibition of the protein expression of class I HDAC 2 and 3 isoforms. Molecular docking analysis of compound 7h revealed that it can prominent binding with the active pocket of the HDAC 2. These finding suggest that the compound 7h can be a promising lead candidate for further investigation in the development of novel anti-cancer drug potentially inhibiting HDACs.

β-Carboline and N-hydroxycinnamamide hybrids as anticancer agents for drug-resistant hepatocellular carcinoma

Ling, Yong,Gao, Wei-Jie,Ling, Changchun,Liu, Ji,Meng, Chi,Qian, Jianqiang,Liu, Siqun,Gan, Huiling,Wu, Hongmei,Tao, Jinhua,Dai, Hong,Zhang, Yanan

, p. 515 - 526 (2019/03/08)

In an effort to develop anticancer agents that may overcome drug resistance, the number one reason in caner death, we have developed a series of novel hybrids of β-carboline and N-hydroxycinnamamide as histone deacetylase (HDAC) inhibitors. Most of the hybrids 13a-p showed strong antiproliferative effects with low-micromolar IC50 values against four human cancer cells. The most potent compound of series 13p exhibited high HDAC1/6 inhibitory effects, and also increased the acetylation levels of histone H3, H4 and α-tubulin. Importantly, 13p demonstrated high anticancer potency against drug-sensitive HepG2 and Bel7402 cells and drug-resistant Bel7402/5FU cells. Hybrid 13p triggered significant apoptosis by regulating apoptotic relative proteins expression in these Bel7402/5FU cells. Finally, 13p induced a substantial amount of autophagic flux activity by the accretion of the expression of LC3-II and the degeneration of expression of p62 and LC3-I in Bel7402/5FU cells. Overall, 13p is a novel β-carboline/N-hydroxycinnamamide hybrid with significant anticancer potency that warrants further evaluation for the treatment of drug-resistant hepatocellular carcinoma.

Efficient and practical one-pot conversions of n- tosyltetrahydroisoquinolines into isoquinolines and of N-tosyltetrahydro-β- carbolines into β-carbolines through tandem β-elimination and aromatization

Dong, Jing,Shi, Xiao-Xin,Yan, Jing-Jing,Xing, Jing,Zhang, Qiang,Xiao, Sen

experimental part, p. 6987 - 6992 (2011/02/24)

An efficient, practical, and general method for conversions of N-tosyltetrahydroisoquinolines (N-tosyl-THIQs) into isoquinolines and of N-tosyltetrahydro-β-carbolines (N-tosyl-THBCs) into β-carbolines is described. Treatment of N-tosyl-THIQs or N-tosyl-THBCs with base in dimethyl sulfoxide afforded dihydroisoquinolines or dihydro-β-carbolines as intermediates, and these were then oxidized in situ by molecular oxygen to furnish isoquinolines or β-carbolines in good to high yields. Both one-pot conversions occurred through tandem β-elimination and aromatization. An efficient method for conversions ofN-tosyltetrahydroisoquinolines (N-tosyl-THIQs) and N-tosyltetrahydro-β-carbolines (N-tosyl-THBCs) into isoquinolines and β-carbolines is described. Treatment ofN-tosyl-THIQs or N-tosyl-THBCs with base affords dihydroisoquinolines or dihydro-β- carbolines. These can be oxidized in situ by molecular oxygen to furnish isoquinolines or β-carbolines. Copyright

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