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N-(2-nitro-4-methoxyphenyl)anthranilic acid is a chemical with a specific purpose. Lookchem provides you with multiple data and supplier information of this chemical.

92906-29-1

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92906-29-1 Usage

Check Digit Verification of cas no

The CAS Registry Mumber 92906-29-1 includes 8 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 5 digits, 9,2,9,0 and 6 respectively; the second part has 2 digits, 2 and 9 respectively.
Calculate Digit Verification of CAS Registry Number 92906-29:
(7*9)+(6*2)+(5*9)+(4*0)+(3*6)+(2*2)+(1*9)=151
151 % 10 = 1
So 92906-29-1 is a valid CAS Registry Number.

92906-29-1Relevant academic research and scientific papers

Design, synthesis and anticancer activity evaluation of diazepinomicin derivatives

Yu, Yongguo,Wu, Jianbo,Lei, Fan,Chen, Lei,Wan, Weili,Hai, Li,Guan, Mei,Wu, Yong

, p. 369 - 373 (2013/07/26)

A series of diazepinomicin derivatives were synthesized and evaluated in vitro for their growth inhibitory activity against the human carcinoma cell lines. The results indicated the anticancer selectivity of this kind of compounds. Based on the results, preliminary structure-activity relationships were discussed.

Development of potent and selective inhibitors of aldo-keto reductase 1C3 (type 5 17β-hydroxysteroid dehydrogenase) based on N -phenyl-aminobenzoates and their structure-activity relationships

Adeniji, Adegoke O.,Twenter, Barry M.,Byrns, Michael C.,Jin, Yi,Chen, Mo,Winkler, Jeffrey D.,Penning, Trevor M.

, p. 2311 - 2323 (2012/05/04)

Aldo-keto reductase 1C3 (AKR1C3; type 5 17β-hydroxysteroid dehydrogenase) is overexpressed in castration resistant prostate cancer (CRPC) and is implicated in the intratumoral biosynthesis of testosterone and 5α-dihydrotestosterone. Selective AKR1C3 inhibitors are required because compounds should not inhibit the highly related AKR1C1 and AKR1C2 isoforms which are involved in the inactivation of 5α-dihydrotestosterone. NSAIDs, N-phenylanthranilates in particular, are potent but nonselective AKR1C3 inhibitors. Using flufenamic acid, 2-{[3-(trifluoromethyl)phenyl]amino}benzoic acid, as lead compound, five classes of structural analogues were synthesized and evaluated for AKR1C3 inhibitory potency and selectivity. Structure-activity relationship (SAR) studies revealed that a meta-carboxylic acid group relative to the amine conferred pronounced AKR1C3 selectivity without loss of potency, while electron withdrawing groups on the phenylamino B-ring were optimal for AKR1C3 inhibition. Lead compounds did not inhibit COX-1 or COX-2 but blocked the AKR1C3 mediated production of testosterone in LNCaP-AKR1C3 cells. These compounds offer promising leads toward new therapeutics for CRPC.

Antiproliferative and differentiating activities of a novel series of histone deacetylase inhibitors

Binaschi, Monica,Boldetti, Andrea,Gianni, Maurizio,Maggi, Carlo Alberto,Gensini, Martina,Bigioni, Mario,Parlani, Massimo,Giolitti, Alessandro,Fratelli, Maddalena,Valli, Claudia,Terao, Mineko,Garattini, Enrico

scheme or table, p. 411 - 415 (2011/01/13)

Histone deacetylases are promising molecular targets for the development of antitumor agents. A novel series of histone deacetylase inhibitors of the hydroxamic acid type were synthesized for structure-activity studies. Thirteen tricyclic dibenzo-diazepin

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