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S-2-NITROPHENYL-L-CYSTEINE is a chemical compound derived from the amino acid L-cysteine, featuring a nitrophenyl group. It is recognized for its reversible interactions with proteins and other biomolecules, which makes it a valuable asset in biochemical research. Its versatility and significance extend to applications in the pharmaceutical and biotechnology sectors, particularly in the study of protein interactions, enzyme kinetics, and the synthesis of various chemical compounds. Additionally, it has demonstrated potential in drug development and disease treatment by targeting specific biological pathways.

60115-45-9

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60115-45-9 Usage

Uses

Used in Biochemical Research:
S-2-NITROPHENYL-L-CYSTEINE is used as a research tool for studying protein interactions and enzyme kinetics due to its ability to engage in reversible reactions with proteins and other biomolecules.
Used in Chemical Synthesis:
In the field of chemical synthesis, S-2-NITROPHENYL-L-CYSTEINE is utilized as a building block for the creation of various chemical compounds, contributing to the development of new substances with potential applications.
Used in Drug Development:
S-2-NITROPHENYL-L-CYSTEINE is employed as a potential candidate in drug development, leveraging its capacity to target specific biological pathways, which could be instrumental in the treatment of various diseases.
Used in Disease Treatment:
S-2-NITROPHENYL-L-CYSTEINE is also considered for use in disease treatment, as its interactions with biological systems may offer therapeutic benefits in addressing specific medical conditions.

Check Digit Verification of cas no

The CAS Registry Mumber 60115-45-9 includes 8 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 5 digits, 6,0,1,1 and 5 respectively; the second part has 2 digits, 4 and 5 respectively.
Calculate Digit Verification of CAS Registry Number 60115-45:
(7*6)+(6*0)+(5*1)+(4*1)+(3*5)+(2*4)+(1*5)=79
79 % 10 = 9
So 60115-45-9 is a valid CAS Registry Number.
InChI:InChI=1/C9H10N2O4S/c10-6(9(12)13)5-16-8-4-2-1-3-7(8)11(14)15/h1-4,6H,5,10H2,(H,12,13)/t6-/m0/s1

60115-45-9SDS

SAFETY DATA SHEETS

According to Globally Harmonized System of Classification and Labelling of Chemicals (GHS) - Sixth revised edition

Version: 1.0

Creation Date: Aug 16, 2017

Revision Date: Aug 16, 2017

1.Identification

1.1 GHS Product identifier

Product name S-2-NITROPHENYL-L-CYSTEINE

1.2 Other means of identification

Product number -
Other names S-o-nitrophenyl-L-cysteine

1.3 Recommended use of the chemical and restrictions on use

Identified uses For industry use only.
Uses advised against no data available

1.4 Supplier's details

1.5 Emergency phone number

Emergency phone number -
Service hours Monday to Friday, 9am-5pm (Standard time zone: UTC/GMT +8 hours).

More Details:60115-45-9 SDS

60115-45-9Relevant academic research and scientific papers

Design, synthesis, and structure-activity relationships of macrocyclic hydroxamic acids that inhibit tumor necrosis factor α release in vitro and in vivo

Xue,Voss,Nelson,Duan,Cherney,Jacobson,He,Roderick,Chen,Corbett,Wang,Meyer,Kennedy,Degrado,Hardman,Teleha,Jaffee,Liu,Copeland,Covington,Christ,Trzaskos,Newton,Magolda,Wexler,Decicco

, p. 2636 - 2660 (2007/10/03)

To search for TNF-α (tumor necrosis factor α) converting enzyme (TACE) inhibitors, we designed a new class of macrocyclic hydroxamic acids by linking the P1 and P2′ residues of acyclic anti-succinate-based hydroxamic acids. A variety of residues including amide, carbamate, alkyl, sulfonamido, Boc-amino, and amino were found to be suitable P1 P1-P2′ linkers. With an N-methylamide at P3′, the 13-16-membered macrocycles prepared exhibited low micromolar activities in the inhibition of TNF-α release from LPS-stimulated human whole blood. Further elaboration in the P3′-P4′ area using the cyclophane and cyclic carbamate templates led to the identification of a number of potent analogues with IC50 values of ≤0.2 μM in whole blood assay (WBA). Although the P3′ area can accommodate a broad array of structurally diversified functional groups including polar residues, hydrophobic residues, and amino and carboxylic acid moieties, in both the cyclophane series and the cyclic carbamate series, a glycine residue at P3′ was identified as a critical structural component to achieve both good in vitro potency and good oral activity. With a glycine residue at P3′, an N-methylamide at P4′ provided the best cyclophane analogue, SL422 (WBA IC50 = 0.22 μM, LPS-mouse ED50 = 15 mg/kg, po), whereas a morpholinylamide at P4′ afforded the most potent and most orally active cyclic carbamate analogue, SP057 (WBAIC50 = 0.067 μM, LPS-mouse ED50 = 2.3 mg/kg, po). Further profiling for SL422 and SP057 showed that these macrocyclic compounds are potent TACE inhibitors, with Ki values of 12 and 4.2 nM in the porcine TACE assay, and are broad-spectrum MMP inhibitors. Pharmacokinetic studies in beagle dogs revealed that SL422 and SP057 are orally bioavailable, with oral bioavailabilities of 11% and 23%, respectively.

S-Aryl cysteine S,S-dioxides as inhibitors of mammalian kynureninase

Drysdale, Martin J.,Reinhard, John F.

, p. 133 - 138 (2007/10/03)

A series of 2-amino-S-aryl cysteine S,S-dioxides have been synthesised and shown to inhibit kynureninase an important enzyme in the biosynthesis of the known excitotoxic moiety quinolinic acid. The most potent of these, 2-amino-5-methyl-S-phenyl cysteine S,S-dioxide 6d, inhibits interferon-γ induced synthesis of quinolinic acid in human macrophages.

Detoxification Metabolism of o-Dinitrobenzene by the Yeast Issatchenkia orientalis

Tamaki, Hisanori,Kumagai, Hidehiko,Shimada, Yoshimi,Kashima, Takanori,Obata, Hiroshi,et al

, p. 951 - 956 (2007/10/02)

Culture medium containing 200 μM o-dinitrobenzene suppressed cell growth of Issatchenkia orientalis for about 24 hr and after a lag phase, the cells began to increase with concomitant metabolism of o-dinitrobenzene by glutathione conjugation.The resulting

Angiotensin converting enzyme inhibitors: 1,5-Benzothiazepine derivatives

Slade,Stanton,Ben-David,Mazzenga

, p. 1517 - 1521 (2007/10/02)

The synthesis of chiral 1,5-benzothiazepines 2a-c, 14a-c, 15c, and 16a prepared from cysteine is described. In vitro inhibition of angiotensin converting enzyme (ACE) is reported for each compound. Compound 2c was the most potent in vitro having an ICsub

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