CommentaryFlii Control: Balancing Migration and Adhesion
-
Add time:08/24/2019 Source:sciencedirect.com
Wound healing in the skin requires a compromise between adhesion and migration. Both processes include modulation of the cytoskeleton, cell-surface receptors, and receptor ligands., In this issue, Kopecki et al. demonstrate that overexpression of Flii, an actin-remodeling protein, impedes wound healing but inhibits hemidesmosome formation. In contrast, Flii deficiency results in enhanced wound healing while promoting hemidesmosome assembly. We discuss potential mechanisms that could explain how this unique gelsolin family member might regulate both stable keratinocyte adhesion and motility.
We also recommend Trading Suppliers and Manufacturers of fliI protein (cas 138414-68-3). Pls Click Website Link as below: cas 138414-68-3 suppliers
Prev:Polymorphisms of FLII implicate gene expressions and growth traits in Chinese cattle
Next:Oligomerization of the Bacterial Flagellar ATPase FliI is Controlled by its Extreme N-terminal Region) - 【Back】【Close 】【Print】【Add to favorite 】
- Related Information
- Original ArticleRegeneration of Hair Follicles Is Modulated by Flightless I (Flii) in a Rodent Vibrissa Model08/28/2019
- Interaction of FliI, a component of the flagellar export apparatus, with flagellin and hook protein08/27/2019
- Full length articleFlightless-I (FliI) is a potential negative regulator of the Toll pathway in Litopenaeus vannamei08/26/2019
- Oligomerization of the Bacterial Flagellar ATPase FliI is Controlled by its Extreme N-terminal Region08/25/2019
- Polymorphisms of FLII implicate gene expressions and growth traits in Chinese cattle08/23/2019
-
Health and Chemical more >