Welcome to LookChem.com Sign In|Join Free
  • or
1-ISOPROPOXY-4-NITROBENZENE is a chemical compound characterized by the molecular formula C9H11NO3. It is an organic compound that features a nitro group attached to a benzene ring, with an isopropoxy group as part of its structure. This yellow crystalline solid has a molecular weight of 181.19 g/mol and a melting point of 36-37°C. Due to its potential for causing skin and eye irritation, it is classified as a hazardous chemical that requires careful handling.

26455-31-2

Post Buying Request

26455-31-2 Suppliers

Recommended suppliers

  • Product
  • FOB Price
  • Min.Order
  • Supply Ability
  • Supplier
  • Contact Supplier

26455-31-2 Usage

Uses

Used in Pharmaceutical Industry:
1-ISOPROPOXY-4-NITROBENZENE is used as a building block for the synthesis of various pharmaceuticals. Its unique structure allows it to be a key component in the development of new drugs, contributing to the advancement of medicinal chemistry.
Used in Agrochemical Industry:
In the agrochemical sector, 1-ISOPROPOXY-4-NITROBENZENE is utilized as a precursor in the production of various agrochemicals. Its role in this industry is crucial for the synthesis of compounds that help protect crops and enhance agricultural productivity.
Used in Polymer Industry:
1-ISOPROPOXY-4-NITROBENZENE is employed as a monomer in the manufacture of polymers. Its chemical properties make it suitable for the creation of polymers with specific characteristics, such as improved strength or resistance to certain conditions.
Used in Dye Industry:
1-ISOPROPOXY-4-NITROBENZENE is also used in the dye industry, where it serves as a starting material for the synthesis of various dyes. Its presence in dye formulations contributes to the colorfastness and stability of the dyes in different applications.

Check Digit Verification of cas no

The CAS Registry Mumber 26455-31-2 includes 8 digits separated into 3 groups by hyphens. The first part of the number,starting from the left, has 5 digits, 2,6,4,5 and 5 respectively; the second part has 2 digits, 3 and 1 respectively.
Calculate Digit Verification of CAS Registry Number 26455-31:
(7*2)+(6*6)+(5*4)+(4*5)+(3*5)+(2*3)+(1*1)=112
112 % 10 = 2
So 26455-31-2 is a valid CAS Registry Number.
InChI:InChI=1/C9H11NO3/c1-7(2)13-9-5-3-8(4-6-9)10(11)12/h3-7H,1-2H3

26455-31-2Relevant academic research and scientific papers

Crystallography-guided discovery of carbazole-based retinoic acid-related orphan receptor gamma-t (RORγt) modulators: insights into different protein behaviors with “short” and “long” inverse agonists

Yu, Ming-cheng,Yang, Feng,Ding, Xiao-yu,Sun, Nan-nan,Jiang, Zheng-yuan,Huang, Ya-fei,Yan, Yu-rong,Zhu, Chen,Xie, Qiong,Chen, Zhi-feng,Guo, Si-qi,Jiang, Hua-liang,Chen, Kai-xian,Luo, Cheng,Luo, Xiao-min,Chen, Shi-jie,Wang, Yong-hui

, p. 1524 - 1534 (2020/12/01)

A series of 6-substituted carbazole-based retinoic acid-related orphan receptor gamma-t (RORγt) modulators were discovered through 6-position modification guided by insights from the crystallographic profiles of the “short” inverse agonist 6. With the increase in the size of the 6-position substituents, the “short” inverse agonist 6 first reversed its function to agonists and then to “long” inverse agonists. The cocrystal structures of RORγt complexed with the representative “short” inverse agonist 6 (PDB: 6LOB), the agonist 7d (PDB: 6LOA) and the “long” inverse agonist 7h (PDB: 6LO9) were revealed by X-ray analysis. However, minor differences were found in the binding modes of “short” inverse agonist 6 and “long” inverse agonist 7h. To further reveal the molecular mechanisms of different RORγt inverse agonists, we performed molecular dynamics simulations and found that “short” or “long” inverse agonists led to different behaviors of helixes H11, H11’, and H12 of RORγt. The “short” inverse agonist 6 destabilizes H11’ and dislocates H12, while the “long” inverse agonist 7h separates H11 and unwinds H12. The results indicate that the two types of inverse agonists may behave differently in downstream signaling, which may help identify novel inverse agonists with different regulatory mechanisms.

Aryl Ether Syntheses via Aromatic Substitution Proceeding under Mild Conditions

Ando, Shin,Tsuzaki, Marina,Ishizuka, Tadao

, p. 11181 - 11189 (2020/10/12)

In this study, mild conditions for aromatic substitutions during the syntheses of aryl ethers were developed. In the reaction conditions, the choices of solvent, base, and the sequence for the addition of the reagents proved important. A wide variety of alcohols were used directly as nucleophiles and smoothly reacted with aryl chlorides that possessed either a nitro or a cyano group at either the ortho- or para-position. Controlled experiments we performed suggested that the reaction underwent a charge-transfer process mediated by a combination of DMF and tert-BuOK.

Rationally Designed Polypharmacology: α-Helix Mimetics as Dual Inhibitors of the Oncoproteins Mcl-1 and HDM2

Conlon, Ivie L.,Drennen, Brandon,Lanning, Maryanna E.,Hughes, Samuel,Rothhaas, Rebecca,Wilder, Paul T.,MacKerell, Alexander D.,Fletcher, Steven

, p. 1691 - 1698 (2020/07/04)

Protein–protein interactions (PPIs), many of which are dominated by α-helical recognition domains, play key roles in many essential cellular processes, and the dysregulation of these interactions can cause detrimental effects. For instance, aberrant PPIs involving the Bcl-2 protein family can lead to several diseases including cancer, neurodegenerative diseases, and diabetes. Interactions between Bcl-2 pro-life proteins, such as Mcl-1, and pro-death proteins, such as Bim, regulate the intrinsic pathway of apoptosis. p53, a tumor-suppressor protein, also has a pivotal role in apoptosis and is negatively regulated by its E3 ubiquitin ligase HDM2. Both Mcl-1 and HDM2 are upregulated in numerous cancers, and, interestingly, there is crosstalk between both protein pathways. Recently, synergy has been observed between Mcl-1 and HDM2 inhibitors. Towards the development of new anticancer drugs, we herein describe a polypharmacology approach for the dual inhibition of Mcl-1 and HDM2 by employing three densely functionalized isoxazoles, pyrazoles, and thiazoles as mimetics of key α-helical domains of their partner proteins.

Chromogenic nitrophenolate-based substrates for light-driven hybrid P450 BM3 enzyme assay

Lam, Quan,Cortez, Alejandro,Nguyen, Thanh Truc,Kato, Mallory,Cheruzel, Lionel

, p. 86 - 91 (2016/06/08)

The incorporation of a p-nitrophenoxy moiety in substrates has enabled the development of colorimetric assays to rapidly screen for O-demethylation activity of P450 enzymes. For the light-driven hybrid P450 BM3 enzymes, where a Ru(II) photosensitizer powers the enzyme upon visible light irradiation, we have investigated a family of p-nitrophenoxy derivatives as useful chromogenic substrates compatible with the light-driven approach. The validation of this assay and its adaptability to a 96-well plate format will enable the screening of the next generation of hybrid P450 BM3 enzymes towards C-H bond functionalization of non-natural substrates.

INHIBITORS OF BRUTON'S TYROSINE KINASE

-

Page/Page column 48; 50; 53; 72, (2015/06/25)

This application discloses compounds according to generic Formula (I): wherein all variables are defined as described herein, which inhibit Btk. The compounds disclosed herein are useful to modulate the activity of Btk and treat diseases associated with excessive Btk activity. The compounds are useful for the treatment of oncological, auto-immune, and inflammatory diseases caused by aberrant B-cell activation. Also disclosed are compositions containing compounds of Formula I and at least one carrier, diluent or excipient.

Design, syntheses, and characterization of piperazine based chemokine receptor CCR5 antagonists as anti prostate cancer agents

Arnatt, Christopher K.,Adams, Joanna L.,Zhang, Zhu,Haney, Kendra M.,Li, Guo,Zhang, Yan

supporting information, p. 2319 - 2323 (2014/05/20)

Chemokine receptor CCR5 plays an important role in the pro-inflammatory environment that aids in the proliferation of prostate cancer cells. Previously, a series of CCR5 antagonists containing a piperidine ring core skeleton were designed based upon the proposed CCR5 antagonist pharmacophore from molecular modeling studies. The developed CCR5 antagonists were able to antagonize CCR5 at a micromolar level and inhibit the proliferation of metastatic prostate cancer cell lines. In order to further explore the structure-activity-relationship of the pharmacophore identified, the molecular scaffold was expanded to contain a piperazine ring as the core. A number of compounds that were synthesized showed promising anti prostate cancer activity and reasonable cytotoxicity profiles based on the biological characterization.

Bis(μ-iodo)bis[(-)-sparteine]-dicopper: A versatile catalyst for direct O-Arylation and O-Alkylation of phenols and aliphatic alcohols with haloarenes

Satyanarayana, Ponnam,Maheswaran, Hariharasarma,Kantam, Mannepalli Lakshmi,Bhargava, Suresh

supporting information; experimental part, p. 788 - 790 (2011/08/22)

The easy to prepare dimeric bis(μ-iodo)bis[(-)-sparteine]- dicopper ([CuI{(-)-spa}]2 complex) is shown to be versatile catalyst for O-arylation and O-alkylation with various aryl halides with phenols and aliphatic alcohols respectively, including less reactive aryl chlorides, such as chlorobenzene under mild conditions.

Triton B-mediated efficient and convenient alkoxylation of activated aryl and heteroaryl halides

Meshram,Goud, P. Ramesh,Reddy, B. Chennakesava,Kumar, D. Aravind

experimental part, p. 2122 - 2129 (2010/08/13)

A simple and convenient one-pot synthesis of aryl alkyl ethers by the alkoxylation of aryl halides with alcohol in the presence of Triton B as a base is described. The procedure is applicable for a variety of aryl and heteroaryl halides, and yields are very good. The use of a nonmetallic base and solvent-free conditions are important features of the reaction. Copyright Taylor & Francis Group, LLC.

Photochemical generation and lifetimes in water of p-aryloxy- and p-alkoxyphenylnitrenium ions

Ramlall, Pratima,McClelland, Robert A.

, p. 225 - 232 (2007/10/03)

This paper describes product and flash photolysis studies following irradiation in aqueous solution of 4X-C6H4N3 [X = MeO (12a), EtO (12b), PriO (12c), ButO (12d), C6H5O (12e), 4-MeOC6H4O (12f), F, Cl] and 4-methoxy-l naphthyl azide (15). p-Benzoquinone (or 1,4-naphthoquinone) is observed as a product, in yields of 70-90% with 12a-d, 15, 40% with 12e, 26% with 4-F and 15% with 4-Cl. The quinone arises by a pathway whereby the initially-formed singlet arylnitrene is quenched by protonation by a solvent water molecule to form a nitrenium ion. Hydration of this cation at the para position leads through a hemiacetal (or halohydrin) to the quinone imine, whose hydrolysis results in the final quinone product. Three kinetic processes are observed, the nitrenium hydration on the us time scale, the hemiacetal breakdown on the ms time scale, and the imine hydrolysis on the minutes time scale. The nitrenium ions have lifetimes in aqueous solution of 50 ns (4-PhO), 70 ns (4-MeOC6H4O), 370 ns (4-MeO), 550 ns (4-EtO), 1.25 μs (4-PriO), 1.56 μs (4-ButO) and 1.35 μs (4-methoxynaphthyl). A nitrenium transient is not observed with the 4-halophenyl azides, probably because the lifetime is too short for detection with ns laser flash photolysis (LFP). The alkoxyphenylnitrenium ions are argued to be better represented as oxocarbocations derived from O-alkylation of the quinone imine. The 4-ethoxyphenylnitrenium ion is not quenched by 0.01 mol dm-3 2′-deoxyguanosine, so that k2(dG) is less than 2 × 107 mol -1 dm3 s-1. This contrasts with the 4-biphenylylnitrenium ion, which has a similar solvent reactivity, but reacts with k2(dG) = 2 × 109 mol-1 dm3 s-1. The localization of the positive charge in the alkoxy system is a possible explanation behind this difference.

Post a RFQ

Enter 15 to 2000 letters.Word count: 0 letters

Attach files(File Format: Jpeg, Jpg, Gif, Png, PDF, PPT, Zip, Rar,Word or Excel Maximum File Size: 3MB)

1 Customer Service

What can I do for you?
Get Best Price

Get Best Price for 26455-31-2