Destruction of redox and mitochondrial dynamics co-contributes to programmed cell death in chicken kidney under arsenite or/and copper (II) exposure
-
Add time:07/29/2019 Source:sciencedirect.com
BackgroundSub-chronic arsenic (arsenite) exposure-induced oxidative toxicity leads to adverse effects in various organ systems, especially the kidney. Copper sulphate (Cu2+), known for its extensive uses in agriculture, has also been reported to have pro-oxidation properties. Both of these two potential toxic elements can bio-accumulate through food chain, thus endangering human health. However, their interaction study in the kidney is scanty.
We also recommend Trading Suppliers and Manufacturers of COPPER (II) ARSENITE (cas 10290-12-7). Pls Click Website Link as below: cas 10290-12-7 suppliers
Prev:Copper (II) and/or arsenite-induced oxidative stress cascades apoptosis and autophagy in the skeletal muscles of chicken
Next:Sodium arsenite effect on Vitis vinifera L. Physiology) - 【Back】【Close 】【Print】【Add to favorite 】
- Related Information
- Oxidative stress-induced skeletal muscle injury involves in NF-κB/p53-activated immunosuppression and apoptosis response in copper (II) or/and arsenite-exposed chicken07/31/2019
- Sodium arsenite effect on Vitis vinifera L. Physiology07/30/2019
- Copper (II) and/or arsenite-induced oxidative stress cascades apoptosis and autophagy in the skeletal muscles of chicken07/28/2019
- Effect of copper(II) species on the photooxidation of arsenite under UV-C irradiation07/27/2019
-
Health and Chemical more >


