Biology ContributionMITHRAMYCIN A (cas 18378-89-7) Enhances Tumor Sensitivity to Mitotic Catastrophe Resulting From DNA Damage
-
Add time:08/29/2019 Source:sciencedirect.com
PurposeSpecificity protein 1 (SP1) is involved in the transcription of several genes implicated in tumor maintenance. We investigated the effects of mithramycin A (MTA), an inhibitor of SP1 DNA binding, on radiation response.
We also recommend Trading Suppliers and Manufacturers of MITHRAMYCIN A (cas 18378-89-7). Pls Click Website Link as below: cas 18378-89-7 suppliers
Prev:Synthesis of 5-substituted and 5,6-disubstituted furo[2,3-d]pyrimidines from 2-methylthio-4,6-pyrimidindione and bifunctional electrophiles
Next:Plenary ArticleThe anticancer antibiotic mithramycin-A inhibits TRPV1 expression in dorsal root ganglion neurons) - 【Back】【Close 】【Print】【Add to favorite 】
- Related Information
- Association of a Zn2 + containing metallo β-lactamase with the anticancer antibiotic mithramycin09/07/2019
- Dimerization and DNA recognition rules of MITHRAMYCIN A (cas 18378-89-7)nd its analogues09/06/2019
- Cytotoxic effects of mithramycin DIG-MSK can depend on the rise of autophagy09/05/2019
- Original ArticleMITHRAMYCIN A (cas 18378-89-7) sensitizes therapy-resistant breast cancer stem cells toward genotoxic drug doxorubicin09/04/2019
- The activity of a novel MITHRAMYCIN A (cas 18378-89-7)nalog is related to its binding to DNA, cellular accumulation, and inhibition of Sp1-driven gene transcription09/03/2019
- Quantitative determination of mithramycin in human plasma by a novel, sensitive ultra-HPLC–MS/MS method for clinical pharmacokinetic application09/02/2019
- E1AF promotes MITHRAMYCIN A (cas 18378-89-7)-induced Huh-7 cell apoptosis depending on its DNA-binding domain09/01/2019
- Mithramycin08/31/2019
- Plenary ArticleThe anticancer antibiotic mithramycin-A inhibits TRPV1 expression in dorsal root ganglion neurons08/30/2019
