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potent toxin and tumor promoter of the BHT derivatives
investigated to date, provides additional support for the
proposed route of BHT bioactivation through BHTOH-
QM, and may serve as an additional tool for probing
mechanisms of tumor promotion in mouse lung.
Ack n ow led gm en t. The work reported here was
supported by USPHS Grants CA41248 (J .A.T.) and
CA33497 (A.M.M.) from the National Cancer Institute.
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