Advanced Glycation End Products Stimulate Angiotensinogen Production in Renal Proximal Tubular Cells
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Add time:08/08/2019 Source:sciencedirect.com
ABSTRACTBackgroundElevated advanced glycation end products (AGE) in diabetes mellitus (DM) are implicated in the progression of DM-associated tissue injury, including diabetic nephropathy. The intrarenal renin-angiotensin system, in particular augmentation of angiotensinogen (AGT) in proximal tubular cells (PTC), plays a crucial role in the development of diabetic nephropathy. This study investigated hypothesis that AGE stimulates AGT production in PTC.
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