Hereditary C1 inhibitor deficiency is associated with high spontaneous amidase activity
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Add time:09/10/2019 Source:sciencedirect.com
BackgroundAngioedema diagnosis classically targets the complement system (via C1 inhibitor (C1Inh) function and antigenic C4 level) and contact phase activation (via amidase activity). Bradykinin is responsible for angioedema attacks and is produced from contact phase activation secondary to failed C1Inh control.
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