Journal of Medicinal Chemistry p. 1873 - 1888 (2021)
Update date:2022-08-02
Topics:
Koriyama, Yuji
Hori, Akihiro
Ito, Hisanori
Yonezawa, Shuji
Baba, Yoshiyasu
Tanimoto, Norihiko
Ueno, Tatsuhiko
Yamamoto, Shiho
Yamamoto, Takahiko
Asada, Naoya
Morimoto, Kenji
Einaru, Shunsuke
Sakai, Katsunori
Kanazu, Takushi
Matsuda, Akihiro
Yamaguchi, Yoshitaka
Oguma, Takuya
Timmers, Maarten
Tritsmans, Luc
Kusakabe, Ken-Ichi
Kato, Akira
Sakaguchi, Gaku
Accumulation of amyloid β peptides (Aβ) is thought to be one of the causal factors of Alzheimer's disease (AD). The aspartyl protease β-site amyloid precursor protein cleaving enzyme 1 (BACE1) is the rate-limiting protease for Aβ production, and therefore
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