1068-84-4Relevant articles and documents
Under-flame oxidation of amines and amino acids in an aqueous solution
Nomoto, Shinya,Shimoyama, Akira,Shiraishi, Susumu,Sahara, Denzo
, p. 1851 - 1855 (1996)
Flames from town gas-oxygen, hydrogen-oxygen, and ethylene-oxygen mixtures, when blown against the surface of an aqueous solution of amines and amino acids, induced an oxidation reaction in the aqueous phase, while an acetylene-oxygen flame failed to oxidize the compounds in solution. The hydrogen flame caused direct hydroxylation of the aromatic rings of phenylglycine homologs. The isomeric ratio of o-, m-, and p-hydroxyphenyl- amino acids produced was in accordance with that obtained by using the reaction systems of Fe2+-H2O2-EDTA and Fe2+-ascorbic acid-H2O2-EDTA, which are known to involve a hydroxyl radical as the agent for hydroxylating the aromatic rings. These results strongly suggest that the active species of flame-induced oxidation in an aqueous solution was the hydroxyl radical which was produced in the flames and extracted into the aqueous phase.
OXIDATIVE DEGRADATION OF HYDROXY AMINO ACIDS BY CONTACT GLOW DISCHARGE ELECTROLYSIS
Harada, Kaoru,Terasawa, Jun-ichi,Gunji, Hiromi
, p. 1545 - 1548 (1980)
Oxidative degradation of several hydroxy amino acids was carried out in order to confirm the reaction pathway of the oxidative degradation of β- and γ-amino acids by contact glow discharge electrolysis.The results indicate that the reaction products from hydroxy amino acids support the hypothesis that the oxidative degradation of β- and γ-amino acids pass through hydroxy amino acids.
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Cassatt,J.C.,Wilkins,R.G.
, p. 6045 - 6050 (1968)
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RENIN INHIBITORS IV
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, (2008/06/13)
The invention concerns novel renin-inhibitory peptides which are useful for treating renin-assocated hypertension, hyperaldosteronism, and congestive heart failure. Processes for preparing the peptides, compositions containing them and methods of using them are included. Also included is a diagnostic method which uses the compounds to determine the presence of renin-associated hypertension or hyperaldosteronism.
